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Część badań wskazuje na potencjał neuroprotekcyjny BPC-157 oraz na jego rolę w utrzymaniu homeostazy tlenku azotu (NO) w różnych modelach urazu
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Meanwhile, Gpx1 −/− mice were protected from the high-fat diet-induced insulin resistance by a mechanism related to enhanced oxidation of the PI3K antagonist phosphatase and tensin homolog (Pten) [75]
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