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We observed a rapid degradation of PPARγ protein in the NEDD4 overexpression group, which was ameliorated upon treatment of HK-2 cells with the proteasome inhibitor MG132 (Fig
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Some KA subunits allow Ca 2+ efflux, which in astrocytes can contribute to glutamate vesicular release or activate other Ca 2+ -dependent signaling pathways (see Guerra-Gomes et al., 2018)
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