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Early-stage AMPK activation during AKI may fulfill elevated energy demands and support cellular repair mechanisms ( 4 Specific role of mitochondrial energy metabolic reprogramming in AKI During AKI, metabolic reprogramming in TECs may temporarily compensate for deficits in energy supply, support cellular survival, and partially mitigate tissue damage, thereby facilitating early repair processes
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Retinofugal projections from melanopsin-expressing retinal ganglion cells revealed by intraocular injections of Cre-dependent virus
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