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The induction of ferroptosis in Leydig cells inhibits testosterone synthesis during late puberty.55 ROS accumulation and ferritin heavy chain 1 and GPx4 inhibition may be possible mechanisms by which exposure to T-2 toxin leads to ferroptosis and testicular injury in male mice.20 The available evidence suggests that OS may play a dual role in Leydig cells, potentially stimulating proliferation while ultimately impairing differentiation and steroidogenic function ( Table 2 and Figure 2 )

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