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37): (i) by direct oxidation of a target protein, (ii) by oxidation via a highly reactive sensor protein, (iii) by activation of a target protein upon dissociation of an oxidized inhibitor, (iv) by oxidation of a target protein via a secondary product generated through e.g
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demonstrated that SLC31A1 plays a crucial role in cisplatin-induced AKI by triggering cuproptosis through mitochondrial dysfunction [11]
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