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Under oxidative conditions, NRF2 dissociates from KEAP1 and translocates to the nucleus, where it binds to and activates the antioxidant response element (ARE) in a variety of target genes
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first, to disseminate already resistant bacteria and genes within and between humans and animals, and second, as a source and facilitator for the evolution of AMR (Stanton et al., 2022)
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